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Congestive Cardiomyopathy in Association with the Acquired Immunodeficiency Syndrome

List of authors.
  • Ira S. Cohen, M.D.,
  • David W. Anderson, M.D., Ph.D.,
  • Renu Virmani, M.D.,
  • Bernard M. Reen, M.D.,
  • Abe M. Macher, M.D.,
  • Joel Sennesh, M.D.,
  • Paul DiLorenzo, M.D.,
  • and Robert R. Redfield, M.D.

Infection with human immunodeficiency virus (HIV, or HTLV-III/LAV) results in global immune dysfunction and leads to a breakdown in the ability of the host to mount an immune response, thereby facilitating unremitting infections by predominantly opportunistic pathogens,1 2 3 4 with or without Kaposi's sarcoma. These secondary events result in organ-system damage and in some cases in organ failure. To date, serious organ-system failure has been documented in the pulmonary, gastrointestinal, renal, and central nervous systems.4 5 6 Clinical cardiac involvement has received little attention except as a site of spread of Kaposi's sarcoma.7 , 8 Recently, we observed three fatal cases of the acquired immunodeficiency syndrome . . .

Funding and Disclosures

The opinions and assertions contained herein are the private views of the authors and are not to be construed as official or reflecting the views of the Department of the Army or the Department of Defense.

Author Affiliations

From the Cardiology Service, Department of Medicine, and the Department of Pathology, Walter Reed Army Medical Center, the Armed Forces Institute of Pathology, and the Walter Reed Army Institute of Research, Washington, D.C.; the Fairfax Hospital, Falls Church, Va.; and the Department of Medicine, Uniformed Services University of the Health Sciences, Bethesda, Md. Address reprint requests to Dr. Cohen at the Non-Invasive Cardiology Laboratory, Walter Reed Army Medical Center, Washington, DC 20307–5001.

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